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Condition Guide

New Treatments & Clinical Trials for Chronic Myeloid Leukemia

Last updated May 2026Data from ClinicalTrials.gov271 active trials
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Chronic myeloid leukemia (CML) is driven almost entirely by a single genetic change — the BCR-ABL fusion gene — that tyrosine kinase inhibitors (TKIs) can control so effectively that most patients now have near-normal life expectancy. Research has shifted toward achieving treatment-free remission and managing the rare patients who resist all TKIs.

What's actually going on in research

Third-generation TKIs like asciminib specifically target the BCR-ABL myristoyl pocket rather than the ATP-binding site, overcoming resistance to earlier drugs including ponatinib. Treatment-free remission — stopping TKIs and maintaining deep molecular response — is now achievable for many patients and is being optimized in trials. Emerging approaches using immune therapies to eradicate residual CML stem cells that hide from TKIs are in early trials.

Asciminib (STAMP inhibitor)

Asciminib targets BCR-ABL through a completely different binding site than prior TKIs, producing responses in patients resistant or intolerant to two or more earlier therapies.

Treatment-free remission

Trials are defining who can safely stop TKI therapy after achieving deep molecular remission and what predicts durable response-free status.

CML stem cell eradication

CML stem cells persist even with deep molecular remission. Trials are testing immunotherapy and combination approaches that may eliminate this reservoir to achieve genuine cure.

What to know before you search

Eligibility depends on CML phase (chronic, accelerated, blast), specific BCR-ABL kinase domain mutations, prior TKI history, and molecular response depth.

What types of trials are currently open

  • TKI optimization trialsComparing different TKIs as initial therapy and testing newer generation drugs in resistant or intolerant patients.
  • Treatment-free remission trialsTesting criteria and monitoring strategies for safely stopping TKI therapy.
  • Resistant CML trialsEvaluating options for T315I and compound mutations resistant to available TKIs.
  • Stem cell eradication trialsTesting immunotherapy combinations to eliminate residual CML stem cells and achieve deeper remissions.
  • Observational studiesTracking long-term cardiovascular effects of TKIs and quality of life on treatment.

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